상세 정보

underline
Metadata Downloads : dc(xml) or Excel
Cited 0 time in scopus ci

Title 

How does SA signaling link the Flg22 responses?

Authors 

So Young YiSuk Yoon Kwon

Publisher 

Landes Bioscience

Issue Date 

2014

Citation 

Plant Signaling & Behavior, vol. 9, no. 11, pp. e972806-e972806

Keywords 

Callose depositionFlg22Flg22-triggered oxidative burstFLS2FRK1NPR1SASA-mediated primingSID2WRKY29

Abstract 

Salicylic acid (SA) has a central role in activating plant resistance to pathogens. SA levels increase in plant tissue following pathogen infection and exogenous SA enhances resistance to a broad range of pathogens. To study the relevance of the SA signaling in the flg22 response, we investigated the responses of SA-related mutants to flg22, a 22-amino acid peptide of the flagellin bacterial protein. We identified SA as an important component of the flg22-triggered oxidative burst, a very early event after flg22 detection, and gene induction, an early event. SA acted partially by enhancing accumulation of FLS2 mRNA. We also provide new evidence that NPR1 play a role in SA-induced priming event that enhances the flg22-triggered oxidative burst, which is correlated with enhancement of the flg22-induced callose deposition. Based on these observations, we conclude that SA signaling is required for early as well as late flg22 responses.

ISSN 

1559-2316

Link 

http://dx.doi.org/10.4161/15592316.2014.972806

Appears in Collections

1. Journal Articles > Journal Articles

Registered Date

2017-04-19


There are no files associated with this item.
qrcode

FusionCharts.
DSpace Software Coptright(c) 2010 MIT and Hewleft-Packard  /  KRIBB-REPOSITORY ( Email:jakim@kribb.re.kr)